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CRIg, a tissue-resident macrophage specific immune checkpoint molecule, promotes immunological tolerance in NOD mice, via a dual role in effector and regulatory T cells
Retrograde fibroblast growth factor 22 (FGF22) signaling regulates insulin-like growth factor 2 (IGF2) expression for activity-dependent synapse stabilization in the mammalian brain
What was the Mg2+ concentration?

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Clarinet (CLA-1), a novel active zone protein required for synaptic vesicle clustering and release
Retrograde fibroblast growth factor 22 (FGF22) signaling regulates insulin-like growth factor 2 (IGF2) expression for activity-dependent synapse stabilization in the mammalian brain

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Gene free methodology for cell fate dynamics during development
What was the Mg2+ concentration?

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CRIg, a tissue-resident macrophage specific immune checkpoint molecule, promotes immunological tolerance in NOD mice, via a dual role in effector and regulatory T cells
What was the Mg2+ concentration?
This is a reply.

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Molecular basis of fatty acid taste in Drosophila
Retrograde fibroblast growth factor 22 (FGF22) signaling regulates insulin-like growth factor 2 (IGF2) expression for activity-dependent synapse stabilization in the mammalian brain
What was the Mg2+ concentration?
Only me

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CRIg, a tissue-resident macrophage specific immune checkpoint molecule, promotes immunological tolerance in NOD mice, via a dual role in effector and regulatory T cells
Retrograde fibroblast growth factor 22 (FGF22) signaling regulates insulin-like growth factor 2 (IGF2) expression for activity-dependent synapse stabilization in the mammalian brain
https://images.pexels.com/photos/707837/pexels-photo-707837.jpeg?w=940&h=650&auto=compress&cs=tinysrgb

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CRIg, a tissue-resident macrophage specific immune checkpoint molecule, promotes immunological tolerance in NOD mice, via a dual role in effector and regulatory T cells
<math xmlns="http://www.w3.org/1998/Math/MathML"><mlongdiv><mn> 12 </mn><mn> 16.5 </mn> <mn> 198 </mn> <msgroup position='1' shift='-1'> <msgroup> <mn> 12</mn> <msline length='2'/> </msgroup> <msgroup> <mn> 78</mn> <mn> 72</mn> <msline length='2'/> <mn> 6.0</mn> <mn> 6.0</mn> </msgroup> <msgroup position='-1'> <!-- extra shift to move to the right of the "." --> <msline length='3'/> <mn> 0</mn> </msgroup> </msgroup> </mlongdiv> </math>
Long division ftw.

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  1. Cell biology
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Retrograde fibroblast growth factor 22 (FGF22) signaling regulates insulin-like growth factor 2 (IGF2) expression for activity-dependent synapse stabilization in the mammalian brain

Akiko Terauchi et al
Feedback signaling between the synapse and nucleus via FGF22 and IGF2 directs the activity-dependent stabilization of presynaptic terminals in the mouse hippocampus.

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  1. Cell biology
  2. Epidemiology and global health
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Retrograde fibroblast growth factor 22 (FGF22) signaling regulates insulin-like growth factor 2 (IGF2) expression for activity-dependent synapse stabilization in the mammalian brain

Akiko Terauchi et al
Feedback signaling between the synapse and nucleus via FGF22 and IGF2 directs the activity-dependent stabilization of presynaptic terminals in the mouse hippocampus.

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  1. Cell biology
  2. Epidemiology and global health
Meaningful alt text here please.

Retrograde fibroblast growth factor 22 (FGF22) signaling regulates insulin-like growth factor 2 (IGF2) expression for activity-dependent synapse stabilization in the mammalian brain

Akiko Terauchi et al
Feedback signaling between the synapse and nucleus via FGF22 and IGF2 directs the activity-dependent stabilization of presynaptic terminals in the mouse hippocampus.

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An image.

eLife Lens search

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Multicellular life, potato blight and Hepatitis B

Episode 24
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The eLife Sciences 2015 Annual Report

The centre of the stage at eLife in 2015 was occupied by a new human ancestor 'Homo naledi' discovered by scientists in an extraordinary find in South Africa and published in eLife in two stunning papers in September. That these scientists chose to publish such ground-breaking findings in eLife is testament both to the journal's growing significance, and to the steady cultural shift towards greater transparency and collaboration in science, which lie at the heart of eLife's mission.